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Haufen von Aufrichtigkeit Debatte thapsigargin er stress mechanism Veränderbar erweitern Verbessern

Frontiers | STING, the Endoplasmic Reticulum, and Mitochondria: Is Three a  Crowd or a Conversation?
Frontiers | STING, the Endoplasmic Reticulum, and Mitochondria: Is Three a Crowd or a Conversation?

Unfolded Protein Response (UPR) and Endoplasmic Reticulum (ER) Stress FAQs
Unfolded Protein Response (UPR) and Endoplasmic Reticulum (ER) Stress FAQs

Effects of endoplasmic reticulum stress on apoptosis induction in  radioresistant macrophages
Effects of endoplasmic reticulum stress on apoptosis induction in radioresistant macrophages

Endoplasmic Reticulum Stress Mediates Vascular Smooth Muscle Cell  Calcification via Increased Release of Grp78 (Glucose-Regulated Protein, 78  kDa)-Loaded Extracellular Vesicles | Arteriosclerosis, Thrombosis, and  Vascular Biology
Endoplasmic Reticulum Stress Mediates Vascular Smooth Muscle Cell Calcification via Increased Release of Grp78 (Glucose-Regulated Protein, 78 kDa)-Loaded Extracellular Vesicles | Arteriosclerosis, Thrombosis, and Vascular Biology

Thapsigargin - Wikipedia
Thapsigargin - Wikipedia

Thapsigargin-induced severe ER stress stimulates release of... | Download  Scientific Diagram
Thapsigargin-induced severe ER stress stimulates release of... | Download Scientific Diagram

IJMS | Free Full-Text | Thapsigargin—From Traditional Medicine to  Anticancer Drug
IJMS | Free Full-Text | Thapsigargin—From Traditional Medicine to Anticancer Drug

What is Thapsigargin? Frequently Asked Questions
What is Thapsigargin? Frequently Asked Questions

MITOL prevents ER stress‐induced apoptosis by IRE1α ubiquitylation at ER–mitochondria  contact sites | The EMBO Journal
MITOL prevents ER stress‐induced apoptosis by IRE1α ubiquitylation at ER–mitochondria contact sites | The EMBO Journal

ER stress-induced cell death proceeds independently of the TRAIL-R2  signaling axis in pancreatic β cells | Cell Death Discovery
ER stress-induced cell death proceeds independently of the TRAIL-R2 signaling axis in pancreatic β cells | Cell Death Discovery

Neuronal Apoptosis Induced by Endoplasmic Reticulum Stress Is Regulated by  ATF4–CHOP-Mediated Induction of the Bcl-2 Homology 3-Only Member PUMA |  Journal of Neuroscience
Neuronal Apoptosis Induced by Endoplasmic Reticulum Stress Is Regulated by ATF4–CHOP-Mediated Induction of the Bcl-2 Homology 3-Only Member PUMA | Journal of Neuroscience

Tunicamycin specifically aggravates ER stress and overcomes chemoresistance  in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation |  Journal of Experimental & Clinical Cancer Research | Full Text
Tunicamycin specifically aggravates ER stress and overcomes chemoresistance in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation | Journal of Experimental & Clinical Cancer Research | Full Text

Phenotypic Discovery of Neuroprotective Agents by Regulation of Tau  Proteostasis via Stress‐Responsive Activation of PERK Signaling - Shin -  2021 - Angewandte Chemie International Edition - Wiley Online Library
Phenotypic Discovery of Neuroprotective Agents by Regulation of Tau Proteostasis via Stress‐Responsive Activation of PERK Signaling - Shin - 2021 - Angewandte Chemie International Edition - Wiley Online Library

Frontiers | Mechanism of Endoplasmic Reticulum Stress in Cerebral Ischemia
Frontiers | Mechanism of Endoplasmic Reticulum Stress in Cerebral Ischemia

Endoplasmic Reticulum Stress Induces G2 Cell-Cycle Arrest via mRNA  Translation of the p53 Isoform p53/47 - ScienceDirect
Endoplasmic Reticulum Stress Induces G2 Cell-Cycle Arrest via mRNA Translation of the p53 Isoform p53/47 - ScienceDirect

Frontiers | The Cross-Links of Endoplasmic Reticulum Stress, Autophagy, and  Neurodegeneration in Parkinson's Disease
Frontiers | The Cross-Links of Endoplasmic Reticulum Stress, Autophagy, and Neurodegeneration in Parkinson's Disease

Tunicamycin specifically aggravates ER stress and overcomes chemoresistance  in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation |  Journal of Experimental & Clinical Cancer Research | Full Text
Tunicamycin specifically aggravates ER stress and overcomes chemoresistance in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation | Journal of Experimental & Clinical Cancer Research | Full Text

Cell death induced by the ER stressor thapsigargin involves death receptor  5, a non-autophagic function of MAP1LC3B, and distinct contributions from  unfolded protein response components | Cell Communication and Signaling |  Full Text
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components | Cell Communication and Signaling | Full Text

Thapsigargin promotes colorectal cancer cell migration through upregulation  of lncRNA MALAT1
Thapsigargin promotes colorectal cancer cell migration through upregulation of lncRNA MALAT1

Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in  Pharmacological Sciences
Thapsigargin: key to new host-directed coronavirus antivirals?: Trends in Pharmacological Sciences

Endoplasmic reticulum stress signalling – from basic mechanisms to clinical  applications - Almanza - 2019 - The FEBS Journal - Wiley Online Library
Endoplasmic reticulum stress signalling – from basic mechanisms to clinical applications - Almanza - 2019 - The FEBS Journal - Wiley Online Library

Targeting endoplasmic reticulum stress in insulin resistance: Trends in  Endocrinology & Metabolism
Targeting endoplasmic reticulum stress in insulin resistance: Trends in Endocrinology & Metabolism

EGF-SubA enhances cytotoxic effects of the ER stress inducer... | Download  Scientific Diagram
EGF-SubA enhances cytotoxic effects of the ER stress inducer... | Download Scientific Diagram

Revealing Protein Aggregates under Thapsigargin-Induced ER Stress Using an  ER-Targeted Thioflavin | ACS Sensors
Revealing Protein Aggregates under Thapsigargin-Induced ER Stress Using an ER-Targeted Thioflavin | ACS Sensors

Pancreatic INS-1 β-Cell Response to Thapsigargin and Rotenone: A  Comparative Proteomics Analysis Uncovers Key Pathways of β-Cell Dysfunction  | Chemical Research in Toxicology
Pancreatic INS-1 β-Cell Response to Thapsigargin and Rotenone: A Comparative Proteomics Analysis Uncovers Key Pathways of β-Cell Dysfunction | Chemical Research in Toxicology

Endoplasmic reticulum stress stimulates the release of extracellular  vesicles carrying danger-associated molecular pattern (DAMP) molecules |  Oncotarget
Endoplasmic reticulum stress stimulates the release of extracellular vesicles carrying danger-associated molecular pattern (DAMP) molecules | Oncotarget